(Last updated : 2019-03-08 07:48:36)
  宮野 佳
   Department     ,
   Position  
■ Present specialized field
Medical biochemistry 
■ BooK
1. 2010   The Nox family of NADPH oxidases that deliberately produce reactive oxygen species., In Free Radical Biology in Digestive Diseases, Front. Gastrointest. Res. 
■ Journal
1. 2018/06 Differential cell surface recruitment of the superoxide-producing NADPH oxidases Nox1, Nox2 and Nox5: The role of the small GTPase Sar1. 
2. 2016/12 TRPC3-GEF-H1 axis mediates pressure overload-induced cardiac fibrosis. 
3. 2016/11 TRPC3 positively regulates reactive oxygen species driving maladaptive cardiac remodeling. 
4. 2014/12 DOCK2 and DOCK5 Act Additively in Neutrophils To Regulate Chemotaxis, Superoxide Production, and Extracellular Trap Formation. 
5. 2014/09 Arachidonic Acid Induces Direct Interaction of the p67phox-Rac Complex with the Phagocyte Oxidase Nox2, Leading to Superoxide Production. 
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■ Presentation
1. 2011/09/06 The Nox family NADPH oxidases involved in host defense and signal transduction  
2. 2010/06/06 Role for the N-terminal transmembrane and C-terminal cytosolic moieties of Nox-family NADPH oxidases.  
3. 2010/06/06 Structure and regulation of Nox family NADPH oxidases that deliberately produce reactive oxygen species  
4. 2006/10/16 Direct Involvement of the Small GTPase Rac in Activation of the Superoxide- Producing NADPH Oxidase Nox1.  
5. 2006/06/18 Direct Involvement of the Small GTPase Rac in Activation of the Superoxide- Producing NADPH Oxidase Nox1.