Present specialized field
（Last updated : 2021-09-21 08:12:32）
Present specialized field
Immunology, Laboratory animal science, Bacteriology
CD157 Confers Host Resistance to Mycobacterium tuberculosis via TLR2-CD157-PKCzeta-Induced Reactive Oxygen Species Production.
IL-6-PAD4 axis in the earliest phase of arthritis in knock-in gp130F759 mice, a model for rheumatoid arthritis
Development of a new mouse model of nasal hypersensitivity
Involvement of IL-17A-producing TCR gamma delta T cells in late protective immunity against pulmonary Mycobacterium tuberculosis infection.
Interleukin-22-Induced Antimicrobial Phospholipase A2 Group IIA Mediates Protective Innate Immunity of Nonhematopoietic Cells against Listeria monocytogenes.
Anti-apoptotic roles for the mutant p53R248Q through suppression of p53-regulated apoptosis-inducing protein 1 in the RA-derived fibroblast-like synoviocyte cell line MH7A.
A-pro-inflammatory role for A20 and ABIN family proteins in human fibroblast-like synoviocytes in rheumatoid arthritis.
A pro-inflammatory role for A20 and ABIN family proteins in human fibroblast-like synoviocytes in rheumatoid arthritis.
Essential role of IL-17A in the formation of a mycobacterial infection-induced granuloma in the lung.
Suppressed induction of mycobacterial antigen-specific Th1-type CD4
T cells in the lung after pulmonary mycobacterial infection.
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BST-1/CD157 promotes epithelial damages in DSS-induced colitis.
Lack of BST-1/CD157 ameliorated fibroblastic changes of arthritis in GP130F759 through down regulation of IL-6 production by synovial fibroblasts.
IL-6-PAD4 axis in the preclinical state of the synovium in gp130F759
-interaction between fibroblast-like synoviocytes and neutrophils-
IL-6-PAD4 axis: IL-6 dependent PAD4 production in neutrophils at the earliest phase of arthritis in gp130F759.
Toll-like receptor-induced Ab production from marginal zone B cells is negatively regulated by ADP-ribosyl cyclase BST-1/CD157
macrophage-like synoviocytes in gp130F759, a mouse model for rheumatoid arthritis
BST-1/CD157 negatively regulates marginal zone B cell survival and Ab production induced with Toll-like receptor stimulation
exacerbated the arthritis in gp130F759.
Elongation of the small intestine and enlargement of the mesenteric lymph nodes in Bst1Cd38 double knockout mice.
A novel function of ADP-ribosyl cyclases to regulate the length of the small intestine revealed in CD38/CD157 double knockout mice
Accelerated onset of arthritis by systemic infection of Mycoplasma fermentans in the knock-in mice gp130F759
BST-1/CD157 on B cells negatively regulates Toll-like receptor signaling
Involvement of IL-17A-producing TCR gd
T cells in late protective immunity against pulmonary
Padi4 expression by neutrophils in the synovium appears in the earliest phase of arthritis in gp130F759
Lack of microbiota revealed the role for BST-1/CD157 to maintain the homeostasis at mucosa-MLN interface
Systemic infection with
facilitates development of arthritis in the knock-in mice gp130F759 through the action of neutrophils.
Enhanced TI-1 antigen responses in BST-1/CD157 knock-out mice of C57BL/6J background
Critical roles of neutrophils for development of arthritis in the knock-in mouse gp130F759 induced by systemic infection of
High density of TNFR2 on fibroblast-like synoviocyte derived from rheumatoid arthritis provides the sensitivity to cell death.
Increase of neutrophils in the synovia precedes the onset of arthritis induced by infection with
in the knock-in mouse gp130F759.
facilitates the progress of arthritis in the knock-in mouse gp130F759
Disease-promoting roles for eosinophils in antigen-induced arthritis.
Increase of PADI4 in the joints and anti-CCP antibody in the sera in a murine model for rheumatoid arthritis before the clinical onset of arthritis
Eosinophils are increased in the synovium of gp130F759 before the clinical onset of srthritis.
gene expression in the joints and serum anti-CCP antibody at very early stage of arthritis in the knock-in mouse gp130F759.
Increase of inflammatory cells and expression of
in the joints of the murine rheumatoid arthritis model gp130F759 before the clinical onset of arthritis
The role of IL-17A producing TCR γδ T cells in the granuloma formation in the lung during mycobacterial infection.
Essential role of interleukin-17A in the formation of a mycobacterial infection-induced granuloma in the lung.
Role of IL-17 producing gd T cells in the granuloma formation in the lung during
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Japan College of Rheumatology
The Molecular Biology Society of Japan
© 2008 Kawasaki Medical School