（Last updated : 2020-07-03 15:56:10）
Gene expression profiles for macrophage in tissues in response to different exercise training protocols in senescence mice
intake improves the reduced lipopolysaccharide-induced TNF-α production that occurs upon exhaustive exercise in mice.
Special Issue "Exercise and Inflammation"
Reduction of real-time imaging of M1 macrophage chemotaxis toward damaged muscle cells is PI3K-dependent
Benefits of post-stress immunosuppression: a viewpoint from exercise immunology
Reduction of voluntary physical activity in mice induced by toll-like receptor 7 agonist R-848.
Resistance training prevents muscle fibrosis and atrophy via down-regulation of C1q-induced Wnt signaling in senescent mice.
Altered gut microbiota by voluntary exercise induces a high physical activity in high-fat diet mice.
Palmitoleic acid induces the cardiac mitochondrial membrane permeability transition despite the presence of L-carnitine
Exhaustive exercise increases the TNF-a production in response to flagellin via the upregulation of toll-like receptor 5 in the large intestine in mice.
Relationship between macrophage differentiation and the chemotactic activity toward damaged myoblast cells
The suppression of tumor necrosis factor-alpha production in response to pathogen stimulation by strenuous exercise and underlying mechanisms
Exercise training attenuates hepatic inflammation, fibrosis and macrophage infiltration during diet induced-obesity in mice.
Interferon-beta, but not tumor necrosis factor-alpha, production in response to poly I:C is maintained despite exhaustive exercise in mice.
Protective action of L-carnitine on cardiac mitochondrial function and structure against fatty acid stress.
L-carnitine is essential to beta-oxidation of quarried fatty acid from mitochondrial membrane by PLA(2)
L-Carnitine suppresses oleic acid-induced membrane permeability transition of mitochondria.
Display 5 items
Impact of wheel running on behavioral anxiety and gut microbiota in mice fed with a high-fat-diet
Regulation of gut microbiota-induced metabolites by voluntary exercise in a high-fat-diet fed mice
The complementary effect of dietary-fiber intake and voluntary exercise on the gut microbiota in high-fat-diet induced obese mice
Relationship between physical activity and gut microbiota in exercised TLR5 deficient mice
Changes in the gut microbiota of highly trained distance runners during a summer training camp in a hot environment.
Effects of voluntary exercise on plasma and urinary metabolites and gut microbiota in a high-fat-diet fed mice.
Effects of wheel running on blood-brain-barrier permeability in high-fat-diet fed mice.
Voluntary wheel running accelerates PHGG-induced gut environmental improvement in high-fat-diet fed mice.
Voluntary wheel running influences on dynamics of gut microbiota F/B ratio in TLR5 deficient mice.
The influence of gut microbiota on Tnf-alpha expression in skeletal muscle of antibiotic treated mice
The preventive effect of voluntary physical exercise on mental disorders in high-fat-diet-mice.
Gut microflora diversity is induced by resistance training in aged mice, but its acquired diversity might not be contributed with the high physical performance.
Exploring beneficial impact of physical activity from body compositions of fecal occult blood-positive patients with colon polyps in Japan.
Aerobic and resistance training-induced attenuation of chronic inflammation involves functional change of macrophages in various tissues of senescent mice.
Reduced serum C1q by resistance training is related to the prevention of sarcopenia.
Chronic responses of inflammation and macrphpage function to exercise training in various tissues of senescence mice.
Altered gut microbiota by voluntary exercise in donor mice may contribute to high physical activity in recipient mice.
Gut microbiota in exercise trained Toll-like receptor 5 deficient mice inhibits fatty liver and hepatic inflammation.
Voluntary exercise attenuates obesity and systemic inflammation by alteration of gut microbiota in TLR5-deficient mice.
The gut microbiota of TLR5-deficient mice display elevated voluntary physical activity.
The adventures of macrophages toward the damaged skeletal muscle
Flagellin receptor TLR5-deficient mice display elevated voluntary physical activity associated with changes in gut microbiota.
Tumor necrosis factor-alpha production in response to pathogen stimulation via TLRs by strenuous exercise and underlying mechanisms.
Voluntary physical exercise affects metabolic syndrome by alteration of gut microbiota in TLR5-deficient mice.
Flagellin-induced TNF-alpha production is enhanced by exhaustive exercise-induced catecholamine via beta-adrengic receptor.
Real-time imaging of macrophage chemotaxis toward damaged muscle cells is PI3K-dependent.
L-Carnitine induces protection of mitochondria, and that might contribute to inhibit fatty acid-induced arrhythmias of heart.
Relationship between macrophage differentiation and the chemotactic activity toward damaged muscle cells.
The effect of voluntary exercise training on LPS-induced TNF-alpha concentration after exhaustive exercise.
Display 5 items
© 2008 Kawasaki Medical School